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Header of aldosterone

Examples of Aldosterone


12 examples:

High aldosterone levels cause heart damage independently of its well-known effect on blood pressure.
(Nature Communications, )

The renin–angiotensin–aldosterone system (RAAS) regulates blood pressure homeostasis and vascular injury and repair responses.
(Nature Reviews Endocrinology, )

Hyperkalemia is high potassium in the blood, and may occur as a side effect of taking kidney disease medications called Renin Angiotensin Aldosterone System inhibitors.
(Science Daily - News, )

Unfortunately, this triple renin–angiotensin–aldosterone system blockade was associated with worsening of renal function and increases in cardiac events and mortality.
(Nature Reviews Nephrology, )

New research has identified somatic mutations that lead to increased aldosterone production in aldosterone-producing cell clusters (APCC), which are found in normal adrenal glands.
(Nature Reviews Endocrinology, )

Induction of diabetes in wild-type mice has revealed increased levels of angiotensin coverting enzyme 2 (ACE2), which is involved in regulation of the renin–angiotensin–aldosterone system.
(Nature Reviews Nephrology, )

Blockade of the renin–angiotensin–aldosterone system (RAAS) slows the progression of many forms of kidney disease, but whether this therapy is beneficial in kidney transplant recipients is unclear.
(Nature Reviews Nephrology, )

If proven safe and effective for long-term use, these therapies might be administered together with intensive renin–angiotensin–aldosterone blockade to reduce adverse effects and renal and cardiovascular risk.
(Nature Reviews Nephrology, )

But trial investigators and heart failure experts believe it is too early to dismiss hope that spironolactone and other aldosterone antagonists-- including Pfizer's Inspra (eplerenone)-- may eventually be found to work in this population.
(Forbes - Business, )

A new study finds that inhibition of the renin–angiotensin–aldosterone system (RAAS) during focal segmental glomerulosclerosis (FSGS) augments the ability of cells of renin lineage (CoRL) to proliferate and differentiate into podocytes.
(Nature Reviews Nephrology, )

As decreased tissue oxygenation might contribute to acute and chronic kidney disease (CKD), this tool has been examined in patients with CKD. Indeed, the benefits of renin–angiotensin–aldosterone system inhibitors might be partly conferred by increased oxygenation.
(Nature Reviews Nephrology, )

A late-breaking clinical trial, known as the Treatment of Preserved Cardiac Function Heart Failure with an Aldosterone Antagonist trial, demonstrates that spironolactone did not reduce the primary outcome of cardiovascular death, heart failure hospitalization, nor surviving a cardiac arrest in patients with heart failure and preserved ejection fraction.
(Science Daily - News, )

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